| Geno
|
Mag
|
Summary
|
| (T;T)
|
0
|
common in complete genomics
|
| GWAS snp
|
| PMID
|
[PMID 19060906 ]
|
| Trait
|
LDL cholesterol
|
| Title
|
Common variants at 30 loci contribute to polygenic dyslipidemia
|
| Risk Allele
|
C
|
| P-val
|
2E-8
|
| Odds Ratio
|
0.05 [-0.03-0.13] SD decrease
|
| GWAS snp
|
| PMID
|
[PMID 20864672]
|
| Trait
|
|
| Title
|
Genetic Variants Influencing Circulating Lipid Levels and Risk of Coronary Artery Disease
|
| Risk Allele
|
T
|
| P-val
|
1E-11
|
| Odds Ratio
|
0.05 [0.04-0.06] unit increase
|
| GWAS snp
|
| PMID
|
[PMID 20686565]
|
| Trait
|
|
| Title
|
Biological, clinical and population relevance of 95 loci for blood lipids.
|
| Risk Allele
|
C
|
| P-val
|
2E-29
|
| Odds Ratio
|
7.8300 None
|
[PMID 19656773] A polygenic basis for four classical Fredrickson hyperlipoproteinemia phenotypes that are characterized by hypertriglyceridemia.
[PMID 19951432] Analysis of recently identified dyslipidemia alleles reveals two loci that contribute to risk for carotid artery disease.
[PMID 23119086] Variants Identified in a GWAS Meta-Analysis for Blood Lipids Are Associated with the Lipid Response to Fenofibrate
[PMID 23092954] SHAVE: shrinkage estimator measured for multiple visits increases power in GWAS of quantitative traits.
[PMID 25951451] Replication Study in a Japanese Population to Evaluate the Association between 10 SNP Loci, Identified in European Genome-Wide Association Studies, and Type 2 Diabetes
[PMID 30275878] Direct and indirect genetic effects on triglycerides through omics and correlated phenotypes.
]
